Study on a Radant lens Phase Shifter Cell Radant透镜式移相器单元的研究
OBJECTIVE: To investigate the possible mechanism of anticancer activity of the extracts from Inonotus obliquus and its association with the phase of cell cycle. 目的:探讨桦褐孔菌提取物对肿瘤细胞周期的影响及其抗癌的作用机制。
The Preliminary Study of Glucose and Serum Insulin and C-peptid in Acute Pancreatitis; Early Phase Packed Cell Volume 、 Blood Sugar Change Predicting Value to Acute Pancreatitis 急性胰腺炎患者血糖、血清胰岛素及C肽的变化及意义的初步探讨红细胞压积血糖变化对急性胰腺炎预测
Compared with that in the model group, AI decreased, LI and proportion of G 2/ S phase cell were increased in JWD treated group. 健胃汤组与模型组比较,细胞凋亡减少而增殖增加,细胞周期中的G2/M期(有丝分裂期)细胞比率增高。
The DNA index, the incidence of aneuploidy and S phase cell population showed a tendency to increase with the increase of malignancy grade and T classification. 随着肿瘤的增大和恶性程度的增加,其异倍体率、DNA指数和S期细胞比例值也相应增加。
Pravastatin could arrest HT-29 and Ls-174-T cells in the G0/ G1 phase of cell cycle. However, apoptosis in HT-29 and Ls-174-T cells was not obvious. Pravastatin inhibited COX-2 protein expression. 结果体外普伐他汀可抑制HT29和Ls174T细胞增殖,各处理组内G0/G1期细胞增多,但诱导凋亡不明显,体外普伐他汀可减少HT-29和Ls174T细胞株COX2蛋白的表达。
It can induce the expression of cyclin dependent kinase inhibitors ( CDKIs), that may suppress cyclin and cyclin dependent kinases ( CDKs), so it makes cell stagnate some phase in cell cycle. 通过诱导细胞周期素依赖性蛋白激酶抑制物(cki)表达,抑制细胞周期素(cy-clin)和细胞周期素依赖性蛋白激酶(cdk)表达使细胞周期停滞于某一时期。
Conclusion Pravastatin inhibited HT-29 and Ls-174-T colorectal cancer cell growth in vitro, which may be explained by its inhibitory effect on COX-2 protein expression and arresting HT-29 and Ls-174-T cells at G0/ G1 phase of cell cycle. 结论体外普伐他汀对HT-29和Ls174T细胞增殖有抑制作用,该作用可能与使细胞生长阻滞于G0/G1期及抑制COX2蛋白表达有关。
The proportion of cells in S phase of cell cycle was approximately 11% – 23%. 处于细胞周期中S期的细胞比例大约为11% ̄23%。
These results suggest that both PGs and LPA may play an important role in the transition from G0/ G1 phase to S phase of cell cycle of rat cardiac fibroblasts and in the conformation and the development of myocardial hypertrophy through growth regulation of cardiac fibroblasts. 上述研究结果提示:PGs和LPA对乳鼠心脏成纤维细胞G1期至S期的转换有重要的调节作用,并可能通过调节心脏成纤维细胞的生长影响心肌肥厚的形成和发展。
Radiobiology has confirmed that there is much difference between different cell cycle phase in cell radiosensitivity. 处于分裂周期不同时相的细胞其放射敏感性有很大的差异,放射生物学研究已证实,细胞周期中不同时相的细胞放射敏感性有很大差别,G2后期和M期放射敏感性最高。
Conclusion Nimesulide can inhibit the proliferation of Eca-109 cells, cause G_0/ G_1 phase cell cycle arrest and induce apoptosis. 结论尼美舒利可抑制食管癌Eca-109细胞增殖,使细胞周期受阻于G0/G1期并诱导细胞凋亡。
G2+ M phase cell was obviously increased from day 7, which suggested the cell was in the phase of mitosis. G2+M期从d7开始明显增加,表明此时表皮细胞已进入细胞分裂期。
FCM shows that cells are accumulated in S phase in cell cycle; the expression of p21 and cyclin E increase but cyclin A reduces. 流式细胞仪(FCM)检测显示S期细胞积累,p21的表达增加和CyclinA表达减少,而CyclinE的表达增加。
Regulation of G_1 Phase Cell Cycle Regulation Factors in Differentiation and Apoptosis of Matrine Induced Hepatoma Cells 苦参碱诱导人肝癌细胞分化、凋亡时对G1细胞周期调节因子的调控
The percentages of S phase cell nucleus were calculated by image cytometry/ DNA quantitative analysis with CAS 200 System. CAS200图像分析系统DNA定量测定方法统计S期细胞核比例。
Results: Compared with normal control group, AI 、 LI and proportion of all phase cell in cell cycle obviously increased in model group; 结果:模型组胃粘膜细胞AI及LI比正常对照组明显增多,细胞周期中各时相比率明显增加;
The proliferation of the human hepatocellular carcinoma cells was arrested at G1 phase of cell cycles. 肝癌细胞的生长阻滞于G1期。
The transfected cells proliferated actively and S phase of cell cycle was prolonged. 流式细胞仪检测转染后细胞增生活跃,S期明显延长。
S phase cell gradually decreased, while G2/ M phase cell did not obviously change. 细胞周期大部分阻滞于G0/G1期,S期细胞逐渐减少,G2/M期细胞变化不大。
The critical phase of cell differentiation occurs when the death programme is being run. 细胞分化的临界期就处于死亡程序执行中的某个阶段。
Cyclin D1 is a member of cell cycle regulating protein and plays an important role in the check point of G1/ S phase, and its overexpression can accelerate the translation from G1 to S phase and cell proliferation. 细胞周期素D1(cyClinD1)是细胞周期调节蛋白的一员,在G1/S期调控点发挥重要作用;其过表达可加速细胞的G1→S期转换和恶性增殖。
The Effects of Endogenous Photodynamic Therapy on Human Colon Carcinoma Cell Transfected with Plasmids Containing Cytosine Deaminase Gene and G_1 Phase Cell Cycle Checkpoints 内源性光动力学结合CD基因转染增强杀伤结肠癌细胞作用以及对细胞G1期关卡因子的影响以及不确定性来源的内生性等问题。内源性光动力疗法抑制人结肠癌裸鼠种植瘤的实验研究
Degeneration of the nucleus pulposus cells are in G1 phase arrested, the cell can not enter S phase, cell mitosis is inhibited. 退变髓核细胞表现为G1期阻滞,使细胞不能进入S期,细胞有丝分裂受到抑制。
BrdU is a analogous of nucleotide which can be incorporated into DNA chain in S phase of cell cycle. BrdU是一种核苷类似物,在细胞周期的S期可参入DNA链。
Senescent cells showed significant S phase cell cycle arrest and the increase of SA-β-Gal activity. 早衰细胞出现明显的S期细胞周期阻滞,SA-β-Gal活性明显增高。